Direct URL citations appear in the printed text and are provided in the HTML and PDF versions of this article on the journal's Website ( This is an open access article distributed under the Creative Commons Attribution License 4.0 (CCBY), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Supplemental digital content is available for this article. The datasets generated during and/or analyzed during the current study are available from the corresponding author on reasonable request.
The authors declare that they have no competing interests, and all authors should confirm its accuracy. The program does not process any patient's personal data and will not cause any patient hurt. The current analysis does not require ethical approval, because our integrated bioinformatics analysis only collects uploaded data information from the GEO database search.
81673829) and Young Scientists Training Program of Beijing University of Chinese Medicine. The study was financially supported by National Natural Science Foundation of China (Grant Nos. Identification and analysis of key genes associated with acute myocardial infarction by integrated bioinformatics methods. How to cite this article: Guo S, Wu J, Zhou W, Liu X, Liu Y, Zhang J, Jia S, Li J, Wang H. ∗Correspondence: Jiarui Wu, Department of Clinical Chinese Pharmacy, School of Chinese Materia Medica, Beijing University of Chinese Medicine, Beijing 100102, China (e-mail: ).Ībbreviations: AMI = acute myocardial infarction, BP = biological process, CC = cellular component, CCL20 = C–C motif chemokine 20, CK-MB = creatine kinase myocardial band, cTn = cardiac troponin, CVDs = cardiovascular diseases, CXCL1 = growth-regulated alpha protein, CXCL8 = interleukin-8, DEGs = differentially expressed genes, FCER1G = high affinity immunoglobulin epsilon receptor subunit gamma, FPR2 = N-formyl peptide receptor 2, GEO = gene expression Omnibus, GIMAP4 = GTPase IMAP family member 4, GO = Gene Ontology, ICAM1 = intercellular adhesion molecule 1, IL-1 = interleukin-1, IL1B = interleukin-1 beta, JUN = transcription factor AP-1, KEGG = Kyoto Encyclopedia of Genes and Genomes, MCODE = molecular complex detection, MF = molecular function, MMP9 = matrix metalloproteinase-9, NF-κB = nuclear factor kappa-B, PPBP = platelet basic protein, PPI = protein-protein interaction, PTGS2 = prostaglandin G/H synthase 2, TLR2 = toll-like receptor 2, TLR4 = toll-like receptor 4, TNF = tumor necrosis factor, WHO = World Health Organization. Department of Clinical Chinese Pharmacy, School of Chinese Materia Medica, Beijing University of Chinese Medicine, Beijing 100102, China.
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